Chris Exley, Professor in Bioinorganic Chemistry at Keele University, has recently published a new study that demonstrates that the classical senile plaques observed in the brain of people who died with Alzheimer’s disease (AD), are composed of both aluminum and amyloid-b.

“This is the second study confirming significantly high brain aluminum content in fAD but it is the first to demonstrate an unequivocal association between the location of aluminum and amyloid-β in fAD. It shows that two prominent risk factors in the etiology of AD.”

Titled “Aluminum and Amyloid-β in Familial Alzheimer’s Disease”, this latest finding adds to Exley’s ever growing body of research into the toxicity of Aluminum and its role in human disease including Alzheimer’s disease and Autism Spectrum Disorder.

Frustratingly, research into Al and its connection to AD dates back decades, yet has no mainstream appeal. It’s difficult to prove, and there’s no funding in going against a billion dollar industry.

“Aluminum has no known biological function.”

Despite decades of literally finding aluminum inside the plaques, in the neurofibrillary tangles–it’s still not formally recognized as a cause for AD. Instead, once again research proliferates at the notion of “genetic causes” rather than the very obvious environmental triggers.

Christopher Exley and his team have been studying the toxic effects of aluminum for decades. He is considered “the Aluminum Expert.”

In a 2017 paper he authored, titled “Aluminum Should Now Be Considered a Primary Etiological Factor in Alzheimer’s Disease,” Exley discusses what protects from Alzheimer’s disease, and how those interventions fit within the model of a toxic accumulation of aluminum.

For example, people who exercise and sweat as a result (sweating is a major way our body detoxes) resulted in a slowing down in the progression of Alzheimer’s disease, compared to elderly nursing home patients who didn’t exercise.

“While we do not know the cause of Alzheimer’s disease and we do not have any effective therapies to treat the disease, there are a number of ‘environmental’ indices which are known to influence the incidence and progression of Alzheimer’s disease. For example, the incidence of Alzheimer’s disease is higher in females [20] and the onset and progression of Alzheimer’s disease may be delayed by physical exercise [21]. Aluminum as an etiological factor in Alzheimer’s disease links the two in that perspiration is a major route of excretion of aluminum from the body [22]. In the absence of physical exercise, women produce only half the volume of perspiration as men and so may be predisposed to the retention of aluminum in their tissues. In both sexes, physical exercise can increase the perspiration volume many times and so improve the excretion of aluminum from the body. Could exercise-induced improvements in the excretion of aluminum from the body be significant in the benefits of exercise in Alzheimer’s disease?”

Toxicity of Aluminum on the Brain

Way back in 1965, researchers injected aluminum salts into the brains of rabbits (on a different hypothesis) and were astounded to find the adjuvant produced a severe convulsive state following latent period accompanied by striking neuronal changes throughout the central nervous system (CNS). Further investigation revealed that alum phosphate was responsible for this phenomenon and that the neuronal change consisted primary in a neurofibrillary degeneration.

“In 1975, the US Food and Drug Administration (FDA) granted “GRAS

” status to nine different aluminum compounds, including aluminum hydroxide and aluminum phosphate, two aluminum adjuvants used as ingredients in current vaccines.

Demonstrably, the amount of aluminum in just one hepatitis B vaccine is over 14x the “safe limit” for how much a newborn would receive in one day.” (Think Love Healthy)

1965: “Experimental production of neurofibrillary degeneration. 1. Light microscopic observations”

1978: “Aluminum levels in brain in Alzheimer’s disease.”

1995: “Aluminium detected in senile plaques and neurofibrillary tangles is contained in lipofuscin granules with silicon, probably as aluminosilicate.”

1995: “Accumulation of aluminium and silicon in lipofuscin granules.

2005: “Brain aluminum, magnesium and phosphorus contents of control and Alzheimer-diseased patients.”

2016: “The Identification of Aluminum in Human Brain Tissue Using Lumogallion and Fluorescence Microscopy”

To learn more about the toxic effects of aluminum, please read these posts:

6 Documentaries on Aluminum Toxicity

The Truth About the Aluminum In Your Vaccines

Detox Success Story: Baby Is Seizure Free After Aluminum Detox